Interaction of the Yersinia pestis type III regulatory proteins LcrG and LcrV occurs at a hydrophobic interface: correction

Correction In Figure 5 panel B of this article, we reported what was believed to be a mutant of the Pseudomonas aeruginosa (PAO1) PcrG protein capable of trans-complementing an lcrG mutation. After isolation of PcrG mutants that could trans-complement lcrG, we inadvertently compared our mutated pcrG sequences to the pcrG sequence from P. aeruginosa strain 388 instead of strain PAO1. This error resulted in the incorrect characterization of a PcrG variant described in this paper (PcrG F42L). In fact, the residue at position 42 of PcrG from PAO1 is a leucine, not a phenylalanine. Therefore, we did not have a PcrG mutant, but had a wildtype clone.